By Henri Laborit
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2002. Effects of biotin supplementation on horn structure and fatty acid pattern in the bovine claw under ﬁeld conditions. In Proceedings of the 12th International Symposium on Lameness in Ruminants, Orlando, FL, pp. 263–267. Logue, D. , S. A. Kempson, K. A. Leach, J. E. Offer, and R. E. McGovern. 1998. Pathology of the white line. In Proceedings of the 10th International Symposium on Lameness in Ruminants, Lucerne, Switzerland, pp. 142–145. Midla, L. , K. H. Hoblet, W. P. Weiss, and M. L. Moeschberger.
Laminitis—lesions at the cellular level The pathogenesis of laminitis is believed to be associated with a disturbance in the microcirculation of blood in the corium which leads to breakdown of the dermal– epidermal junction between the wall and P3 . As described earlier, rumen acidosis is considered to be a major predisposing cause of laminitis and presumably mediates its destructive effects through various vasoactive substances (endotoxins, lactate, and possibly histamine) that are released into the blood stream in coincidence with the development of rumen acidosis and the subsequent death of rumen microbes.
10). 7 ml. All three cushions extend from the skin–horn junction at the heel toward the tip of the third phalanx. The abaxial and middle fat pads are shorter than the axial pad. The shorter pads lie superﬁcial to the deep digital ﬂexor tendon and do not reach further distal than the insertion of the tendon. The axial fat pad courses from the axial border of the heel bulb toward the middle of the sole surface, where it ends in the middle third. Recent studies by Swiss researchers have shown that the amount of fat (and thus cushioning capacity) increases with increasing age.
Biologie et structure by Henri Laborit